Treatment for Traumatic Brain Injury
Traumatic brain injury rates continue to rise worldwide every year. There is a growing number of chronic disabilities caused by traumatic brain injury (TBI). People under the age of 40 are most likely to be disabled by this disease worldwide. A TBI can cause behavioral problems, mood swings, cognitive impairments, especially memory loss, attention problems, and executive function problems. A major pathogenic mechanism of TBI is the ischemia and hypoxia of brain tissues that cause parenchymal softening and necrosis. Fortunately, the related death rate has declined over time. However, due to an increase in TBIs, there are increasing numbers of individuals living with significant disabilities as a result of their injuries. In traumatized brains, both cytotoxic and vasogenic edema, as well as venous congestion can increase intracranial volume. As a result, the incompressible brain tissue necrosise. It is possible for the brain injury to have persistent, sometimes lifelong effects.
TBI causes an inflammatory response that includes the production of cytokines and chemokines on site, endothelial activation, microglial activation, and neutrophil, lymphocyte, and monocyte migration to the site of injury. Often, concussions are viewed as mild traumatic brain injuries without any gross structural damage. Even mild traumatic brain injuries may cause mild degrees of axonal damage.
The regeneration of neurons and vascular tissue is thought to play a role in brain recovery after brain injury. Hippocampus and subventricular zones in the adult brain exhibit neurogenesis.
Despite the lack of effective treatment for traumatic brain injury recovery today, therapeutic strategies for TBI recovery have been continuously developed over the past several decades. In the treatment of traumatic brain injury, standard medical and surgical interventions are always important. Survivors of TBI with various disabilities are on the rise. The focus of TBI research needs to shift towards neurorestorative and neurorehabilitative therapies. For TBI victims to recover neurological functions, therapeutic strategies targeting both the white matter and hippocampus may be necessary.
These imaging technologies enable us to visualize how acupuncture stimulates the brain anatomically and functionally. It demonstrates that acupuncture can affect neuronal activity in the brain.
The synthesis, release, and action of neurotransmitters (such as serotonin, dopamine, catecholamine, glutamate, acetylcholine, etc.) and neuropeptides (oxytocin, neuropeptide Y, cholecystokinin, vasoactive intestinal peptide, substance P, calcitonin-related peptide, pituitary adenylate cyclase-activating polypeptide) in the central and peripheral nervous systems are involved. Depending on the disease or needling parameters, acupuncture causes different changes in neurotransmitters and neuropeptides.
Acupuncture works so powerfully because of that principle. With acupuncture, nerves and endocrines can be optimized, sleep can be improved, anxiety and depression can be tolerated more easily, blood pressure and blood sugar can be stabilized, GI function is enhanced, stem cells can be mobilized, and all of these effects are amplified through this network to promote brain nerve regeneration.
